Orthopaedic Nurses Certification Board (ONCB) Metabolic/Inflammatory/Tumors Practice Exam

The typical treatment protocol for acute gout attacks focuses on the rapid relief of pain and inflammation associated with the condition. The use of nonsteroidal anti-inflammatory drugs (NSAIDs), colchicine, and corticosteroids are the most effective approaches in managing these acute episodes.

NSAIDs, such as ibuprofen or naproxen, provide significant anti-inflammatory effects and analgesic properties that help reduce the swelling and pain during an attack. Colchicine is another cornerstone medication for gout; it works by decreasing the inflammation caused by uric acid crystals in the joints. Corticosteroids can also be used to reduce inflammation and are particularly helpful for patients who may not tolerate NSAIDs or have contraindications to their use.

In contrast, rest and hydration while important, do not directly address the underlying inflammation during an acute attack. Although hydration is essential for overall health and can help in the elimination of uric acid, it is not a primary treatment for the acute phase. Long-term antibiotic therapy is not relevant for gout, as gout is caused by the crystallization of uric acid, not by bacteria. Increased physical activity is not advisable during an acute gout attack because it could exacerbate the pain and inflammation.

Thus, the combination of NSAIDs, colch